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CB-839 (MCE, HY-12248) was treated at a concentration of 1 M for 48 h

While IN DFO has been extensively studied in preclinical models of Alzheimers disease and stroke, recent developments have expanded the scope to other chelators such as DFP

doi: 10.4103/0253-7613.132160 171 JuSGeYLiPTianXWangHZhengXet al

However, deletion of MLKL or inhibition of RIPK1 in macrophages does not completely rescue Mtb-infected cells from death, 84,101 suggesting that, although the deficiency of MLKL or RIPK1 can abrogate the canonical necroptosis pathway, upstream TNF/TNFR1-mediated signaling may stimulate the induction of other cell death pathways during Mtb infection