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For exercise performance and VO2 max improvement, the evidence from human exercise studies suggests that MOTS-c's acute rise following high-intensity training is part of the adaptive signal driving mitochondrial biogenesis and cardiovascular remodeling

This mechanism does not broadly stimulate cell death but instead targets a survival dependency characteristic of senescent cells

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But based on the evidence presented so far, there is good reason to believe 5-Amino 1MQ is potentially therapeutic for those disease states

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