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[DOI] [PubMed] [Google Scholar] 23.Veeralakshmanan P, Tham JC, Wright A, et al

However, the pharmacokinetic characteristics, tissue bioaccumulation, and toxicity profiles of arsenic metabolites in vivo following oral administration of ATO have not yet been characterized

Ferroptosis, an iron-dependent form of RCD driven by GSH depletion and GPX4 inactivation, is potently accelerated by copper through multi-pronged mechanisms that converge on iron-dependent lipid peroxidation [61, 62]

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Notably, it has been reported that melatonin MT1 receptors prevent -syninduced ferroptosis in Parkinsons disease through activation of the SIRT1/Nrf2/HO-1/GPX4 pathway [40], which is consistent with our findings and further supports the potential of SIRT1 activation as a strategy to suppress ferroptosis