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This study showed that FA transport dysfunction plays is a critical mechanism in both cardiomyocytic injury and HUA-associated LVR and promoting cytoplasmic FA transportation through pharmacological treatment by LC is a valid strategy to attenuate HUA-associated LVR

In summary, nutritional ketosis may serve as a cardioprotective alternative energy supply in the failing heart and reduce pathological ventricular remodeling and could serve as a supportive, non-pharmacologic treatment in people with heart failure along current treatments, such as SGLT2 inhibitors (103, 104)

Adipocyte mitochondria use fat for energy production and deterioration of its function has been implicated in abnormal ectopic fat deposition and insulin resistance [13]

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