increased glutathione levels gliomas autophagy

increased glutathione levels gliomas autophagy and Its Metabolic Enzymes in Gliomal Tumor Tissue and the Peritumoral Zone at Different Degrees of Anaplasia Glutathione metabolism-related gene signature predicts

Name: increased glutathione levels gliomas autophagy and Its Metabolic Enzymes in Gliomal Tumor Tissue and the Peritumoral Zone at Different Degrees of Anaplasia Glutathione metabolism-related gene signature predicts
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Glutathione metabolism related gene signature predicts prognosis and treatment response in low grade glioma Aging Targeting ferroptosis opens new avenues in gliomas Role of Glutathione in Cancer: From Mechanisms to Therapies Frontiers ROS regulation in gliomas: implications for treatment strategies Revisiting the potential of regulated cell death in glioma treatment: a focus on autophagy dependent cell death, anoikis, ferroptosis, cuproptosis, pyroptosis, immunogenic cell death, and the crosstalk between them PMC

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Description

The first indication that TRIMs as a family could directly intersect with the autophagy machinery was published in 2014, when TRIM5, TRIM6, TRIM17, TRIM22, and TRIM49 were shown to interact with the autophagy regulators ULK1 and Beclin 1 (Mandell et al., 2014)