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Furthermore, Sun et al., showed that HuSC-Exos protect against kidney ischemia-reperfusion injury by reducing ferroptosis via lncRNA taurine upregulated gene 1, which regulates ACSL4 through serine arginine-rich splicing factor 1 interaction, suggesting a potential acute kidney injury therapy [72, 106] (Table 1)

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In these cells, ROS are generated via mitochondrial respiration and the activity of inducible nitric oxide synthase (iNOS), which converts arginine to nitric oxide (NO) (79)