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a responder may be considered as a subject for which treatment as described herein provides equal to or more than about 2 points, or about 1 standard deviation, in the VABS-II composite scores

Mechanistically, oxidative stress triggers endoplasmic reticulum stress through the activation of the PERK-eIF2-ATF4 and IRE1-XBP1 signaling pathways, as well as through the upregulation of CHOP (C/EBP homologous protein), which promotes apoptotic signaling (75) as shown in Figure 4

Lincoln GA, Clarke IJ, Hut RA, Hazlerigg DG

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