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In addition, immune-associated genes, such as toll-like receptor 2, tenascin (which can interact with TLRs to prompt inflammatory responses 94,95 are significantly upregulated in aposymbiotic branches (Figure S3)

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An elegant mouse model demonstrated that a heterozygous mutation in the KEAP1 gene is sufficient to reduce KEAP1 activity and consequently stabilize NRF2 (Suzuki et al., 2011