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Iron: the hard player in diabetes pathophysiology, Acta Physiologica

It forces a metabolic shift from oxidative phosphorylation to glycolysis and activates established pro-reprogramming signaling pathways, such as YAP/TAZ, which exacerbate inflammatory responses and fibrosis, thereby accelerating adverse ventricular remodeling [49, 101]

Studies demonstrate improvements with tailored nutrition, antioxidant support, gut restoration, and correction of vitamin D, thyroid, or mitochondrial deficits