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Mechanistically, SLC6A14 promotes ferroptosis in UC by enhancing the expression of CCAAT enhancer binding protein (C/EBP) and its binding activity to inhibit PAK6 (83), providing the SLC6A14-C/EBP-PAK6 axis-mediated ferroptosis a potential therapeutic target for colitis

NRF2 therefore accumulates and is free to translocate to the nucleus through the nuclear pore complex (NPC)

Responses of nontransformed human hepatocytes to conditional expression of full-length hepatitis C virus open reading frame

Total RNA from worms was isolated using TRI reagent (Sigma-Aldrich)

Akt also inactivates the pro-apoptotic Bcl-2 family member BAD (Bcl2/Bcl-XL-antagonist causing cell death) by phosphorylation, thus stopping its interaction with and blockade of the pro-survival Bcl-2 family members Bcl-2 and Bcl-XL (Papadia and Hardingham, 2007)