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The activation initiates autophosphorylation at residues Y1349 and Y1356, leading to the formation of docking points for various adaptor molecules (such as GAB1, GRB2, SHC, CRK, PI3K, PLC1, SHP2, and STAT3), which facilitate the coordination of subsequent signaling [3, 15, 20]

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Translating results from the cardiovascular outcomes trials with glucagon-like peptide-1 receptor agonists into clinical practice: recommendations from a Eastern and Southern Europe diabetes expert group

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J Biol Chem 2018, 293 (20), 75227530