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This link between oncogenic KRAS and FOXO1-mediated changes is, to our knowledge, a novel finding with key implications

doi:10.1507/endocrj.ej12-0106 Vakil M, Park S, Broder A

Consulta con un profesional para valorar el origen y el enfoque adecuado

10.1371/journal.pone.0024141 PLoS ONE 11 BogdanC.RllinghoffM

It is therefore assumed that transcription from gdhA P2 serves to enhance gdhA expression when the concentration of -ketoglutarate is low, as during invasive disease, both to enhance the activity of the TCA cycle in the catabolic reaction, and to provide L-glutamate for glutathione biosynthesis in the anabolic reaction