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Activated ATF6 can also initiate related transcriptional procedures to restore endoplasmic reticulum homeostasis, including inducing GRP78 expression, promoting protein chaperone and lipid synthesis, stimulating endoplasmic reticulum degradation, and improving N-glycosylation (58, 59), and ATF6 also induces CHOP expression, leading to UPR-related apoptosis (60, 61)

Assist in detoxification and cellular repair

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Ma, W., Wei, S., Zhang, B., & Li, W

Funding Open Access funding provided by University of Turku (UTU) including Turku University Central Hospital