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Vingerling et al., 1995

These microorganisms of the intestinal tract also reinforce the barrier function of the intestinal lining, decreasing the passage of pathogenic bacteria or antigens from the gut into the bloodstream [3]

Considering that NF-B activity is directly linked to the LPS-induced expression of iNOS and secretion of NO, it can be concluded that AM404 most likely modulates the secretion of NO from LPS-stimulated microglia through its inhibitory action on the NF-B pathway [70,71]