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Acute mitochondrial GPX4 inactivation upregulates DHODH metabolic flux, accelerating CoQ10H 2 synthesis to neutralize lipid radicals, compensate for GPX4 deficiency and inhibit mitochondrial inner membrane ferroptosis (109)

Hormonal changes, diet, stress, and certain medications can exacerbate these factors, leading to the development and persistence of acne

GPx3 is also one of the 25 known selenoproteins in humans, which harbor a unique selenocysteine found in the active site that utilizes glutathione as the primary reducing agent to remove oxidative compounds 31,32

While the exact sequence of events mediated by Dsg-specific antibodies is not fully clear, three pathways for the induction of acantholysis have been proposed: (i) direct interference with Dsg transinteraction (steric hindrance)

Menezes MSS, Almeida CMM