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subtilis strain expressing both of the active rha1 and rha2 genes versus their catalytically dead counterparts (Suppl Fig 3D, Fig 4D)

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This work was supported by grants to CAB from the National Institute of Health NHLBI (R01 HL144556), the Department of Defense (CA180681), the Hyundai Motor Company (Hope on Wheels Scholar grant), and Curing Kids Cancer Foundation (Innovation Award)

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Overexpression of SLC7A11, a transporter protein that inputs cysteine for the biosynthesis of glutathione and causes antioxidant defense, inhibits ferroptosis and promotes tumor growth [64, 95]