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Under physiological conditions, oxidative free radicals generated by the mitochondrial oxidative respiratory chain, endoplasmic reticulum, and NADPH oxidases can be effectively neutralized by NRF2-mediated antioxidant responses

Accordingly, OS in MS should be regarded not merely as a secondary byproduct of inflammation, but also as an active driver of demyelination, axonal injury, and lesion evolution

3.5 = same as 3 + focal expansion of intercellular spaces

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This conclusion was reached due to findings of large levels of glutathione inside the cell and minuscule levels outside the cell when this channel was absent