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Mechanism of drug tolerant persister cancer cells: the landscape and clinical implication for therapy

For example, an acetylation defect is observed in mutant p53 3 KR , which indirectly inhibits cysteine absorption and reduces GSH consumption, leading to lipid peroxidation and ferroptosis (Wang et al., 2016)

This metabolic dysregulation is defined by systemically reduced levels of metabolites such as amino acids and fatty acids, leading to reduced TCA cycle activity and oxidative phosphorylation, which may ultimately be a major contributor to neuropsychiatric symptoms associated with reduced energy, such as fatigue and other cognitive impairments [43, 62]