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Rev Assoc Med Bras 1992 (2013) 59:42934

25(OH)D is biologically inactive and must be converted to its active form, 1,25(OH)2D3, by 1-hydroxylase in the kidneys (82)

gondii clones resistant to auranofin We mutagenized T

GRX1 deficiency exacerbates this process, leading to enhanced -synuclein toxicity and dopaminergic neurodegeneration in C

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