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On the contrary, the HF6+D group did not show a substantial improvement of heme signals (Figure 6A)

10.1523/JNEUROSCI.3153-13.2014 J

J Pediatr (1997) 130(1):13845.10.1016/S0022-3476(97)70323-8 48 BugianiMLamanteaEInvernizziFMoroniIBizziAZevianiMet alEffects of riboflavin in children with complex II deficiency

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The level of these metabolites in plasma, urine, and cerebrospinal fluid (CSF) is typically elevated with ALDH7A1 mutations ( dependent epilepsy. A similar situation is caused by MOCS2 mutations, which lead to molybdenum cofactor deficiency and sulphite accumulation