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This mechanism could play a role in the development of DILI in individuals taking medications that produce drug-related neoantigens, triggering immune-mediated hepatic injury and compromising liver function [57] (Table 3)

Complement activation in arterial and venous thrombosis is mediated by plasmin

In advanced NSCLC patients who previously received treatment, and who present with alterations in the ALK gene, crizotinib was found to be superior to standard chemotherapy

These signaling cascades finally leads to phosphorylating and activating IKK complex which in turn phosphorylates IB exposing it to the proteasomal degradation, thus unmasking NF-B, permitting its nuclear translocation and eventually triggering the transcription of an assortment of pro-inflammatory genes [47]

MMP, Matrix Metalloproteinase