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Moreover, PDE4 inhibitor and statin may act on different pathways involved in CSE-induced EMT, such as ROS, PI3K/Akt, GTP-Rac1, and nuclear -catenin

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In this case, methylglyoxal was shown to disrupt the interaction between the major pore-forming alpha subunit hSlo1 and gamma regulatory subunit LRRC26 of the BK channel to inhibit its function 20

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