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In fact, Brittany herself even contracted bronchitis because of cigarette smoking, a health care provider at Portsmouth Regional Hospital told her

COPD rat models demonstrated that N -acetylcysteine significantly reduced -SMA level, collagen volume fraction, wall thickness/bronchiole diameter, and wall area/total bronchiole area (MA%) in COPD rats

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Although it is not entirely clear whether it is nicotine per se, the major active element in cigarette smoke and the key ingredient of electronic nicotine delivery devices, nicotine itself has now been shown to enhance airway hyperresponsiveness (12, 14, 19, 22, 23)