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Accordingly, we conclude that the observed antitumorigenic effect of targeting TLRs or epithelial specific MyD88/IKK is mainly due to the inhibition of the NF-B pathway, and subsequent changes in the gradient of essential protumor versus anti-tumor cytokines as we and others have previously shown ( in vivo in NSCLC through the upregulation of VEGF and iNOS, thus promoting an angiogenic phenotype ( Macrophages are an important component of the inflammatory TME

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