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References: Alfaiz, A

However, when APAP is ingested in excessive amounts, the capacity of GSH to neutralize NAPQI becomes overwhelmed, leading to depletion of hepatic GSH stores and accumulation of NAPQI, resulting in oxidative stress, mitochondrial dysfunction, and hepatocellular injury, ultimately culminating in acute liver injury and potential liver failure

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Severino R, Ferrari G, Vu KD, Dons F, Salmieri S, Lacroix M

This metabolic pathway is often hijacked by oncogenic drivers such as RAS and MYC, which enhance CHKA expression to meet the demands of rapid proliferation and membrane biogenesis (Kortlever et al., 2017)