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Thus, GSH reduces the pro-viral DNA load in the first period of infection, suggesting that this antioxidant may be useful for improving current antiviral therapies (Palamara et al., 1996)

After resampling into 500,000-case datasets, linear (LDA) and nonlinear (MLP, DLN) models were developed

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Moreover, intracellular GSH buffered NO production at the post-translational level and participates in protecting monocytes from their own oxidative stress without abrogating NO production and endosome acidification