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Again, the mechanisms involved are hypothesized to be the result of in-utero exposure to environmental stressors ( e.g ., less access to medical resources, including suboptimal prenatal care, maternal stress with potential effects on the fetus, early exposures to toxins, infections, or nutritional difficulties) [105, 108, 118-120]

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Mechanism of hydrogen peroxide-induced inhibition of sheep airway cilia

doi: 10.1016/j.medidd.2020.100027

Promoter engineering of AtSOD1 via CRISPR further demonstrated the potential to modulate expression levels in response to environmental cues, enhancing heat tolerance in Arabidopsis