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Discussion Here, we have demonstrated that the endogenous metabolite glycolate works as an enhancer of the antioxidant response in C

For references and more information, see the section on cardiovascular disease in the Resveratrol article

Most current OA animal models, such as the DMM model or chemically induced models, can simulate cartilage damage in the short term but struggle to fully replicate the long-term cumulative multifactorial pathological changes of human chronic OA

this inhibited the expression of auxin-responsive transcription factor ARF, thereby inhibiting the transduction of auxin signals