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Key distinguishing features, such as a low reticulocyte production index (RPI), hypersegmented neutrophils in cases of B12 and folate deficiencies, dysplastic changes in MDS, and macrocytosis linked to liver dysfunction in alcoholism, can aid in differentiating these causes [9]

Nutrients for Optimisation The most important nutrients for the optimisation of the methylation cycle are: Vitamin B12 (as hydroxo- or methylcobalamin) Methylfolate (as 5-MTHF, Quatrefolic) Vitamin B6 (as pyridoxal-5-phosphate) Magnesium Nutrients for Therapy The following substances are administered for genetic mutations: Methylfolate (as 5-MTHF, Quatrefolic) Bypasses the MTHFR mutations Methionine Bypasses problems with MTHFR SAM Bypasses problems with MTHFR and MAT 5-HTP Bypasses BH4 deficiency or TPH mutations Nutrient Combinations for Certain Symptoms The following nutrient combinations are possible for certain symptoms: Nutrients for MTHFR Mutation MTHFR mutation is by far the most common genetic mutation

35 were preclinical (animal studies)

Methionine is an amino acid that acts as a lipotropic agent to prevent excess fat buildup in the liver and the body

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