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For example, midazolam could facilitate the reuptake of excess Glu via EAAT2 and enhance GABA A R activity, inhibiting the release of Glu transporters, thereby effectively alleviating excitotoxicity caused by the upregulation of NMDAR NR1 subunits after ketamine withdrawal(Li et al

PMID: 130 HoseinifarS

doi: 10.1038/s41587-025-02608-w 170 MokBYde MoraesMHZengJBoschDEKotrysAVRaguramAet al

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