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Oxidative stress caused by acrolein is mainly based on NADPH oxidase-dependent ROS formation, as indicated by Rac1 translocation to the membrane in endothelial cells and macrophages, as well as extracellular ROS formation in macrophages in response to acrolein treatment

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[24] The genome encodes four open reading frames (ORFs), two of which produce a single protein due to ribosomal readthrough of a leaky UAG stop codon

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