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Although endothelial activation and redox signaling compose a normal host defense, when this activation is sustained, chronic production of ROS may exceed the capacity of cellular enzymatic and nonenzymatic antioxidant defenses [49], contributing to triggering vascular diseases such as atherosclerosis or peripheral artery disease [102, 103]

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Consequently, our understanding of a plants ability to respond to stress would be significantly enhanced by investigating the pathways responsible for the phenylpropanoid biosynthesis and plant hormone signal transduction