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High levels of glutathione (GSH) are observed in the majority of tumors, contributing to cancer progression and treatment resistance in part by preventing glutathione peroxidase 4dependent (GPX4-dependent) ferroptosis

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Figure 4 shows the integrated network of cell death pathways triggered by Erastin in endometriosis, illustrating how ferroptosis, necroptosis, and pyroptosis are interconnected and modulated by autophagy-dependent processes

Researchers reported that MOTS-c administration significantly enhanced physical performance in mice across various ages, improving exercise tolerance in not just young but also middle-aged and old mice