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Fer-1 alleviates abnormal expressions of tsRNA, with tsRNA-3029b being an effective target, and inhibiting tsRNA-3029b can inhibit ferroptosis and promote neuronal regeneration

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It is related to the binding of selenoprotein-P to the endothelium in sepsis, to the protein extravasation at the early phase of sepsis, and to downregulation of selenoprotein-P liver synthesis [33]

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Cadmium chloride (CdCl 2 2.5H 2 O) and sodium hydrosulfide (NaHS) were used as Cd and H 2 S donors, respectively