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Protection against APAP-induced hepatoxicity by the superoxide dismutase (SOD) mimetic Mito-TEMPO, together with increased injury in partial SOD-deficient mice, emphasizes the importance of superoxide in this process 7,8,9

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The functional consequences of accumulated mtDNA damage are profound, as oxidization, mutation, or deletion of mtDNA molecules can impair the assembly and function of respiratory chain complexes, leading to deficient ATP synthesis and increased ROS production [19]

Each ADC is essentially a combination of a large biologic (the antibody) and a small-molecule drug, and this dual nature means that analytical and quality control teams must characterize both components as well as the conjugated product