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Vaario LM, Fritze H, Spetz P, Heinonsalo J, Hanajik P, Pennanen T

By inducing excessive oxidative stress, NPC1L1 inhibition increased treatment cytotoxicity and sharply decreased the number of persisters, whereas recovering the key substrates of NPC1L1, cholesterol, and vitamin E, markedly compromised lipotoxicity-mediated oxidative stress

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FIGURE 4 (B) Patient-derived xenograft (PDX) zebrafish models, established by transplanting patient tumor cells into embryos, enable real-time monitoring of tumor engraftment, progression, and therapeutic responses, offering a platform for cancer research and personalized drug evaluation

The peptide nature of ipamorelin also confers relevant research design constraints