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Apoptotic signals increase mitochondrial outer membrane permeability, causing the release of specific apoptotic inducers such as Cytc into the cytosol

These formed MSU crystals can stimulate the release of various inflammatory factors, including TNF-, IL-6, and IL-1, which is achieved by activating the NF-B signaling pathway [5]

Glycolytic Breakdown in Inflammatory Effector Cells In normal synovial tissues, glycolysis is the primary pathway promoting mitochondrial substrate oxidation of pyruvate

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Various human epithelial cells express functional toll-like receptors, nod1 and nod2 to produce anti-microbial peptides, but not proinflammatory cytokines