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Lastly, PPAR-alpha ligands may regulate hepatic gluconeogenesis through modulation of the genes for phosphoenolpyruvate carboxykinase, pyruvate carboxylase, and lactate dehydrogenase A [23]

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Estimates for humans, based upon fraction of drug excreted in urine and assuming mostly hepatic metabolism, suggest a higher threshold of 400-500 daltons for favorable biliary vs renal excretion of small organic molecules