autoregulatory control of mitochondrial glutathione homeostasis

autoregulatory control of mitochondrial glutathione homeostasis in Cellular Redox and Disease Manifestation Mitochondria–ER contacts function as an

Name: autoregulatory control of mitochondrial glutathione homeostasis in Cellular Redox and Disease Manifestation Mitochondria–ER contacts function as an
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MitochondriaER contacts function as an iron supply hub Nature Cell Biology Mitochondrial Glutathione: Regulation and Functions Gaetano Calabrese, Bruce Morgan, Jan Riemer, 2017 SLC25A39 identified as a key regulator of hepatocellular carcinoma progression through the mitochondrial ROScytochrome ccaspase signaling axis Cellular & Molecular Biology Letters Springer Nature Link Dual regulation of SLC25A39 by AFG3L2 and iron controls mitochondrial glutathione homeostasis ScienceDirect glutathione and mitochondrial function Autoregulatory control of mitochondrial glutathione homeostasis

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In addition to the relevance of POP conjugation cited previously, hyperglycemia and hyperinsulinemia appear to directly inhibit glutathione synthesis (GCL), and the increased production of reactive oxygen species which accompanies diabetes further depletes GSH levels