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Mechanistically, inhibition of PDGFR ablates FGF-2-recruited perivascular coverage, exposing anti-VEGF agents to inhibit vascular sprouting

Concurrent treatment of AST with FPN modulated the cleaved caspase-3 immunohistochemical staining in the examined tissues, as most cerebral cortical neurons, cerebellar Purkinje, and granule cells exhibited weak to moderate immunoreactivity