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(73) employed transcription activator-like effector nuclease (TALEN) technology to develop Uox knockout mouse models in a pure C57BL/6J background (73)

In a previous study (Heard et al., 2016), we showed that HFD promoted the establishment of ectopic lesions from endometrial fragments lacking expression of the progesterone receptor interacting protein KLF9, in immunocompetent recipient mice

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