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Reducing brain-generated acetaldehyde by silencing catalase or increasing ALDH2 activity in the VTA decreases alcohol-induced-reinforcing effects and reduces voluntary ethanol intake (141)

However, despite low intra-portal insulin levels, low IGF-I and increased fat mass and decreased lean body mass, their glucose tolerance and insulin sensitivity remains unaffected and in fact, is comparable to normal controls (151, 152)

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In subsequent studies, it was reported that GPx-1 could also protect against cell death in response to cytotoxic drugs, such as doxorubicin, and in response to direct ligand-mediated apoptosis, by ligand for CD95 receptor (CD95L) (142, 143)