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Diabetic mice injected with 100 mg/kg EGCG daily for 4 days exhibited decreased resistance to oxidative stress, as indicated by elevated NADPH oxidase levels and reduced expression of Nrf2, HO-1, and HSP90 (237)

Palmitoylation of V5_GSTP1_4XCysAla/Lys103Arg was retained (Fig 6A), and when the palmitoylation (neutravidin) signal was normalized for GSTP1 level, palmitoylation was reduced by only ~20% (Fig 6B)

Our findings suggest that the absence of these enzymes exacerbates ROS accumulation, thereby promoting ferroptosis and contributing to kidney injury

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