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Methylcobalamin increases Erk1/2 and Akt activities through the methylation cycle and promotes nerve regeneration in a rat sciatic nerve injury model

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AST has been shown to influence core pathological mechanisms in neurodegenerative disease (e.g., AD), including the reduction of tau hyperphosphorylation and suppression of BACE1 activity, both of which are key contributors to neurofibrillary tangle formation and A production, respectively (Table 2