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This review integrates current evidence on how brain peptides contribute to AD pathophysiology, summarizes recent progress in peptide-based therapeutic strategies and delivery platforms, and critically examines the remaining barriers to clinical translation, including bloodbrain barrier penetration, metabolic stability, off-target effects, and the need for biomarker-guided patient stratification

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141 Notably, it has been reported that the first extracellular domains of claudin-2 and claudin-4 are a key determinant of paracellular charge selectivity and barrier function, but these domains are not involved in the formation of tight junction strands

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