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Gilbert M, Shaw WJ, Long JR, Nelson K, Drobny GP, Giachelli CM, et al

Recent evidence (PMID 37981461) highlights FOXO1 as a potential biomarker and therapeutic target in NSCLC metabolic regulation, suggesting that targeting FOXO1-mediated pathways could complement existing metabolic therapies

The foundational building block for this process is an essential amino acid called L-tryptophan, which we obtain by consuming protein-rich foods like poultry, eggs, and dairy

A recent study highlighted the AGR2/IGF1 signaling loop as an example of tumorCAF crosstalk: PDAC cells secrete anterior gradient-2 (AGR2), which activates CAFs through the Wnt/-catenin pathway, causing the CAFs to upregulate and secrete insulin-like growth factor 1 (IGF-1)