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In Ang II-induced HUVECs, GA inhibited Extracellular Signal-Regulated Kinase (ERK) phosphorylation, Cyclophilin D (CypD) expression, the interaction of NOX4 and Poldip 2, and lowered mitochondrial ROS levels, resulting in an increase in the mitochondrial membrane potential of the HUVECs cells, thereby alleviating endothelial mitochondrial dysfunction (Sun et al., 2023)

Growth of Candida albicans cells on the physiologically relevant carbon source, lactate, affects their recognition and phagocytosis by immune cells

Post-challenge hyperglycaemia, nitric oxide production and endothelial dysfunction: the putative role of asymmetric dimethylarginine (ADMA)

Alpha-tocopherol: roles in prevention and therapy of human disease

I do eye cream right after serums so they can both soak in together