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However, whether excess hemin exposure from the hemolysis in SCD will affect CBS expression and the generation of GSH in oxidative and ferroptosis stress response has not been investigated

doi: 10.1046/j.1528-1157.44.s10.3.x

The expression level of glutamate decarboxylase 67 (GAD67, the major enzyme responsible for converting glutamate into GABA) was assessed

This buildup triggers pathophysiological alterations, such as oxidative stress, inflammation, and mitochondrial impairment, which collectively lead to neuronal damage and cognitive deterioration (Wang et al., 2020)

Of note, mitochondrial permeability transition pore (mPTP) has been implicated in oxidative stress-induced necrosis as well as ischemia/reperfusion injury 7