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This growth defect is likely attributed to a compromised methionine biosynthesis pathway, as evidenced by a ~ 1.5-fold decrease in the intracellular methionine levels in the Z::metB KD strain compared to the Rv::metB KD strain (Fig

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Acta Neuropsychiatr 2017;29:337-46

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It is also known that during normal brain development, MCs digest OL progenitor cells [93], raising the possibility that activated MCs might also phagocytose mature OLs in SCZ

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Consequently, mtDNA copy number and mRNA levels of critical electron transport chain (ETC) components were reduced, including: ubiquinone oxidoreductase core subunit 2 (ND2, complex I), SDHA (complex II), CYTB (complex III), cytochrome c oxidase I (COXI, complex IV), and ATP5a1 (complex V) (Fig

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B-12 helps the body process fats and proteins efficiently

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