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The release of cytochrome C from the mitochondria leads to production of a caspase-activating complex of cytochrome c, APAF-1, and procaspase-9, triggers the up-regulation of caspase-9, and subsequently increases the expression of caspase-3, followed by the induction of apoptotic events in the cell [44]

236 Advanced glycation end products (AGEs) formation Chronic hyperglycemia promotes the non-enzymatic glycation of proteins, lipids, and nucleic acids, leading to the formation of AGEs

Ageing hallmarks exhibit organ-specific temporal signatures

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